1633.
A Swedish Farmer's Lineage 61
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In the meanwhile, the disease has appeared in the male ancestor's family, but not in the females, and from this fact we can deduct that the male ancestor must have had the propensity and not the female. A sister of the male ancestor must have had the same propensity, because, when a grandchild of hers, married a grandchild of the male ancestor, two of their several children had this serious disease. Both these parents must therefore have been heterozygots, and the propensity must have originated from the male ancestor and his sister, through one of their parents, but not from the female ancestor, or her family.
The first bearer of this propensity for myoklonus epilepsy for which we have any information thus lived in the 17th century. There we lose all trace. It is impossible to determine whether the propensity has originated in Sweden, or was brought here from some other country.
Professor Unverricht in Dorpat was the first to describe the disease in question.
He has observed several cases in one and the same household in Esthonia. It is not only possible, but very probably that these families living in Sweden and Esthonia, had the same root many years ago, for diseases of this type go in
[title to genealogy graphic]Inheritance Scheme Relating to the Family Disease Myoklonus Epilepsy in Blekinge.
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